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Health Risk WARNING: Do You Have Wi-Fi Sickness Symptoms

Physicist Warns of Health Impact Caused by Radio Frequency/Microwave Radiation Produced by Wireless Devices.
Dr. Ronald M Powell, PhD in applied physics from Harvard University has published a paper warning of the health risks associated with wireless devices.
“Simply stated, a worldwide health crisis is emerging and is becoming a hallmark of the 21st Century,” claims Dr. Powell in a recent document warning schools of the danger of WiFi. However his demand for caution is falling on deaf ears and he is not the only one, “the international biomedical research community is trying to warn us; but we, in the USA, are not yet listening.”
The Harvard graduate believes individuals struggle with cognitive dissonance over the issue stating that “genuine usefulness of wireless devices promotes denial of the risks.”
Powell’s voice is not alone, other expert’s in the industry have similar concerns. He proposes that “thousands of peer-reviewed studies published in biomedical research journals have contributed to our understanding of this impact. So many serious biological effects have been found that immediate responsive action is warranted. Further, these biological effects are occurring at levels of radiation far lower than earlier understood.”
There are many detailed health issues that may be cause from wireless devices, all living things are bioelectrical in nature. Powell explains this is why “electrocardiograms and electroencephalograms work. They, of course, measure the tiny electrical signals that operate the heart and the brain. The critical tasks performed by these tiny electrical signals, and so many other electrical signals in all living things, can be disrupted by radiofrequency/microwave radiation.”
Side effects and dangers of Wi-Fi sickness include:
  • sleep disruption
  • headaches
  • fatigue
  • ringing in the ears
  • memory loss
  • dizziness
  • heart arrhythmia
  • DNA damage
  • cancer
  • infertility

Dr. Powel continued to state individuals can not control exposure to these devices and that “even aware individuals cannot control their exposure in any environment shared with others, because the radiation around them, much like second-hand smoke, is forced on them by unaware individuals. Self education and prevention of repercussions for the time being are the best way to prepare, “for now the public will have to protect itself, and that will require public education and action,” documented Powell, Phd ( via therundownlive.com ).

In 2013 Dr. Powell filed a complaint with the FCC calling for a “reassessment of federal communications,” in regards to “commission radiofrequency exposure limits and policies.”

Medical breakthrough: Paraplegic man walks with own legs again

“A man who lost the use of his legs to a spinal cord injury has walked again after scientists rerouted signals from his brain to electrodes on his knees. The 26-year-old American has used a wheelchair for five years after an accident left him paralysed from the waist down. Doctors said he was the first person with paraplegia caused by a spinal injury to walk without relying on robotic limbs that are controlled manually.

The man walked a 3.5-metre course after being fitted with an electrode cap that picks up brain waves and beams them wirelessly to a computer, which decipher the waves as an intention to stand still or walk. The relevant command is then sent to a microcontroller on the man’s belt, and on to nerves that trigger muscles to move the legs.

The patient needed intensive training to generate recognisable walking signals in his brain, and to learn how to use the device to put one foot in front of the other. He also needed extensive physical training to build up the muscle tone in his legs.

“Even after years of paralysis, the brain can still generate robust brain waves that can be harnessed to enable basic walking,” said Dr An Do at the University of California at Irvine, who co-led the proof-of-concept study. “We showed that you can restore intuitive, brain-controlled walking after a complete spinal cord injury. This non-invasive system for leg muscle stimulation is a promising method and is an advance on our current brain-controlled systems, which use virtual reality or a robotic exoskeleton.”

The man learned to produce the right brain signals by moving an avatar around a virtual environment while he was sitting down wearing the cap, which has a built-in electroencephalogram (EEG) that monitors brain waves. When he had made sufficient progress, he practised walking for real while suspended 5cm above the ground, so that he could move his legs freely without having to support his weight. On the patient’s 20th session, he used the system to walk on the ground, helped by a walking frame that prevented him from falling over. Over the 19-week course, he learned better to control the device and so the movement of his legs. Rather than having a precise control of each leg, the patient activates the system with a general concept of walking, Do told Radio 4’s Today programme on Thursday. “It’s not so much that he’s thinking ‘move the right leg and than move the left leg’,” he said. “What happens is that the computer system detects when the brain waves change from a state of not walking into a state of walking.

“When the computer detects that a person is walking, based on these brain waves, it turns on the electrical stimulator, which starts creating muscle contractions in the right leg first, and then the left leg; right leg, left leg. And then it keeps on doing this automatically until he stops thinking about walking, then it shuts it off and keeps him in a standing position. So really he has the control of a general concept of walk or not walk.”

A spinal cord injury severs the nerves that carry signals from the brain to the limbs. The nerves do not grow back and often scar tissue forms at the site of the damage. The proof-of-concept device shows the potential for computerised systems that can read signals from the brain, bypass damaged areas, and feed them back into the healthy nerves that control the muscles for walking.

Researchers at the lab stressed that they had only tested the brain-computer interface on one patient and that many more patients would be needed for trials before they could assess its more general usefulness for people with paraplegia. Details of the study are reported in the Journal of NeuroEngineering and Rehabilitation.

If the device works well in others, the electrode cap could eventually be swapped for a hidden implant that monitors brain signals from beneath the skull.

Zoran Nenadic, a colleague of Do’s at Irvine, said: “We hope that an implant could achieve an even greater level of prosthesis control, because brain waves are recorded with higher quality. In addition, such an implant could deliver sensation back to the brain, enabling the user to feel their legs.”

In their report, the scientists describe how the patient was able to conduct a “light conversation” with the test team while attempting to walk. The scientists have a number of major hurdles to overcome before the device can be used to help paraplegics walk freely. In laboratory tests, the computer got confused when the patient was walking on the ground, potentially because brain signals for walking became mixed up with others for balance and stabilisation.

Another problem is the bulk of the system, which will need to be solved with smaller, implantable devices. Writing in the journal, the researchers say: “The cumbersome nature of the current noninvasive system makes its adoption for restoration of overground walking unlikely ( via theguardian.com ). “This limitation can potentially be addressed by a fully implantable brain-computer interface system, which can be envisioned to employ invasively recorded neural signals.”” copied.

 

Depression Can Physically Alter Your DNA

Article by: Caroline Reid

Depression doesn’t just change your mentality—it also leaves marks on your DNA, report scientists from the Wellcome Trust Centre for Human Genetics (WTCHG). The finding was so surprising that the scientists initially met it with skepticism and admit that they required a substantial amount of convincing to believe it was not merely a coincidence.

As described in Current Biology, the discovery began with an examination of the DNA of over 11,000 people, many of whom had a history of stress-related depression, but healthy controls were also included for comparison. Initially, scientists were searching for a gene associated with an increased risk of depression. Instead, researchers noticed that stress-related depression correlated with an increase in the amount of the cell’s second genome: mitochondrial DNA.

Mitochondria are the powerhouses of the cell: they create energy for cells to flourish. The increase in mitochondrial DNA of people with depression has interesting implications. Mitochondria struggle to create energy for cells as effectively when they’re under stress, so the body needs to compensate and create more and more mitochondria to keep up with the increased energy demand.

Taking this one step further, the scientists then tested the molecular changes in mice that were subject to four weeks of stress. They found not only that the mice exhibited the expected increase in mitochondrial DNA, but also that their telomeres had reduced in length. Telomeres are little caps on the end of our DNA strands that shield our chromosomes from degradation and thus the loss of genetic information. Every time a cell makes a copy of itself, the length of the protective telomere gets a little bit shorter until the cell can no longer divide.

The erosion of telomeres in these stressed mice indicates that stress can decrease one’s life expectancy. But it’s not all doom and gloom, as the researchers also discovered that the changes in both telomere-length and the increase in mitochondrial DNA are largely reversible. After the mice were free from stress, their DNA also recovered.

So why does stress-induced depression, classically considered to be a mood disorder, have these cellular-level effects on the body? There are plenty of causes for stress: a lack of food or a history of abuse, for example. Stress decreases the efficiency of the mitochondria, and as a result the body creates more to protect its metabolism. In some sense, depression may be the body’s response to environmental stress.

This study has exciting potential for future treatment of depression. Since the molecular changes in the DNA are reversible, there is now the possibility to assess how successful treatment is on a molecular level. The possible future indicators for success would be a reduction in the number of mitochondria and restored telomere-length.

Humans may harbor more than 100 genes from other organisms

Article by Sarah C. P. Williams

You’re not completely human, at least when it comes to the genetic material inside your cells. You—and everyone else—may harbor as many as 145 genes that have jumped from bacteria, other single-celled organisms, and viruses and made themselves at home in the human genome. That’s the conclusion of a new study, which provides some of the broadest evidence yet that, throughout evolutionary history, genes from other branches of life have become part of animal cells.

“This means that the tree of life isn’t the stereotypical tree with perfectly branching lineages,” says biologist Alastair Crisp of the University of Cambridge in the United Kingdom, an author of the new paper. “In reality, it’s more like one of those Amazonian strangler figs where the roots are all tangled and crossing back across each other.”

Scientists knew that horizontal gene transfer—the movement of genetic information between organisms other than parent-to-offspring inheritance—is commonplace in bacteria and simple eukaryotes. The process lets the organisms quickly share an antibiotic-resistance set of genes to adapt to an antibiotic, for instance. But whether genes have been horizontally transferred into higher organisms—like primates—has been disputed. Like in bacteria, it’s been proposed that animal cells could integrate foreign genetic material that’s introduced as small fragments of DNA or carried into cells by viruses. But proving that a bit of DNA in the human genome originally came from another organism is tricky.

Crisp and his colleagues analyzed the genome sequences of 40 different animal species, ranging from fruit flies and roundworms to zebrafish, gorillas, and humans. For each gene in the genomes, the scientists searched existing databases to find close matches—both among other animals and among nonanimals, including plants, fungi, bacteria, and viruses. When an animal’s gene more closely matched a gene from a nonanimal than any other animals, the researchers took a closer look, using computational methods to determine whether the initial database search had missed something.

In all, the researchers pinpointed hundreds of genes that appeared to have been transferred from bacteria, archaea, fungi, other microorganisms, and plants to animals, they report online today in Genome Biology. In the case of humans, they found 145 genes that seemed to have jumped from simpler organisms, including 17 that had been reported in the past as possible horizontal gene transfers.

“I think what this shows it that horizontal gene transfer is not just confined to microorganisms but has played a role in the evolution of many animals,” Crisp says, “perhaps even all animals.

The paper doesn’t give any hints as to how the genes—which now play established roles in metabolism, immune responses, and basic biochemistry—may have been transferred or the exact timeline of the jumps, he says. That will take more work.

The findings are critical to understanding evolution, says Hank Seifert, a molecular biologist at the Northwestern University Feinberg School of Medicine in Chicago, Illinois. “This is a very well-done paper. They used all the latest data they could find, all the genomes in the databases,” he says. “It makes it clearer than ever that there has been a history, throughout evolution, of gene transfer between organisms.”

But not all agree that the new evidence is indisputable. “I see little here that is particularly convincing evidence for horizontal gene transfer,” says microbiologist Jonathan Eisen of the University of California, Davis. He doesn’t rule out that horizontal gene transfer between bacteria and animals is possible, but says that there are other explanations for the identified genes being present in only some branches of the evolutionary tree—a gene that existed in a far-off ancestor could have simply been lost in many relatives other than two seemingly unrelated species, for instance. “It is up to [the researchers] to exclude other, more plausible alternatives, and I just do not think they have done that.”

*Correction, 16 March, 12:37 p.m.: The piece has been updated to clarify the fact that bacteria are not eukaryotes.

MEDICAL BREAKTHTOUGH: Gold Plated Nano-Particles seek and destroy Cancer Cells

February 7, 2015 – In a recently published study, researchers from Cornell University detail how they developed gold-plated nano-particles that are able to find and destroy cancer cells.

Comparable to nano-scale Navy Seals, Cornell scientists have merged tiny gold and iron oxide particles to work as a team, then added antibody guides to steer the team through the bloodstream toward colorectal cancer cells. And in a nanosecond, the alloyed allies then kill the bad guys – cancer cells – with absorbed infrared heat.

This scenario is not science fiction – welcome to a medical reality.

“It’s a simple concept. It’s colloidal chemistry. By themselves, gold and iron-oxide alloys are benign and inert, and the infrared light is low-power heating,” said Carl Batt, Cornell’s Liberty Hyde Bailey Professor of Food Science and the senior author on the paper. “But put these inert alloys together, attach an antibody to guide it to the right target, zap it with infrared light and the cancer cells die. The cells only need to be heated up a few degrees to die.”

Batt and his colleagues – Dickson K. Kirui, Ph.D. ’11, a postdoctoral fellow at Houston Methodist Research Institute and the paper’s first author; Ildar Khalidov, radiology, Weill Cornell Medical College; and Yi Wang, biomedical engineering, Cornell.

For cancer therapy, current hyperthermic techniques – applying heat to the whole body – heat up cancer cells and healthy tissue, alike. Thus, healthy tissue tends to get damaged. This study shows that by using gold nano-particles, which amplify the low energy heat source efficiently, cancer cells can be targeted better and heat damage to healthy tissues can be mitigated. By adding the magnetic iron oxide particles to the gold, doctors watching MRI and CT scanners can follow along the trail of this nano-sized crew to its target.

When a near-infrared laser is used, the light penetrates deep into the tissue, heating the nano-particle to about 120 degrees Fahrenheit – an ample temperature to kill many targeted cancer cells. This results in a threefold increase in killing cancer cells and a substantial tumor reduction within 30 days, according to Kirui. “It’s not a complete reduction in the tumor, but doctors can employ other aggressive strategies with success. It also reduces the dosage of highly toxic chemicals and radiation – leading to a better quality of life,” he explained.

The study was funded in part by the Ludwig Institute for Cancer Research, and Kirui was supported by a Sloan Foundation Graduate Fellowship.